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- Inflammation is induced when host cells sense evolutionarily conserved structures on pathogens (pathogen-associated molecular patterns) or endogenous stress signals (danger-associated molecular patterns) through germline-encoded pattern-recognition receptors (PRRs) 3.
www.nature.com/articles/ni.3790
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A coordinated series of common effector mechanisms of inflammation contribute to tissue injury, oxidative stress, remodeling of the extracellular matrix, angiogenesis, and fibrosis in diverse target tissues.
- Molecular and Cellular Mechanisms of Inflammation and Tissue ...
Inflammation is associated with the activation of the innate...
- Inflammation: From Cellular Mechanisms to Immune Cell ...
Acute inflammation is a localized, self-limited innate host...
- Molecular and Cellular Mechanisms of Inflammation and Tissue ...
Inflammation is associated with the activation of the innate immune system. At the site of inflammation, neutrophils appear first, followed by monocytes, which may differentiate into macrophages. The main function of macrophages and immune cells is to remove cell debris and microorganisms.
Jul 19, 2017 · Kastner and colleagues review monogenic inflammatory diseases caused by mechanisms as varied as dysregulated inflammasomes, perturbations in ubiquitination, protein folding or complement...
Dec 1, 2007 · A coordinated series of common effector mechanisms of inflammation contribute to tissue injury, oxidative stress, remodeling of the extracellular matrix, angiogenesis, and fibrosis in diverse target tissues.
Month:Total Views:September 202446August 2024131July 2024109June 2024131- Peter Libby
- 2007
Sep 9, 2020 · Acute inflammation is a localized, self-limited innate host defense mechanism against invading pathogens and tissue injury. Typically, polymorphonuclear neutrophil granulocytes (PMN) are among the first cells to be recruited from the blood to the site of infection or injury.
- János G Filep, Amiram Ariel
- 10.1152/ajpcell.00181.2020
- 2020
- 2020/09/09
Jul 29, 2011 · Inflammatory responses are essential for the maintenance of normal tissue homeostasis. The molecular mechanism of inflammation is quite a complicated process which is initiated by the recognition of specific molecular patterns associated with either infection or tissue injury.
Inflammation provides a unifying pathophys-iological mechanism underlying many chronic diseases, including diabetes, cardiovascular disease, certain can-cers and bowel diseases, arthritides, and osteoporosis. Common pathophysiologic scenarios apply to many of these diseases.
