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Death genes", ced-3 and ced-4
- In 1986, he identified the first "death genes", ced-3 and ced-4. He showed that functional ced-3 and ced-4 genes were a prerequisite for cell death to be executed.
en.wikipedia.org/wiki/H._Robert_Horvitz
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In his early work at MIT, Horvitz continued his work on cell lineage and cell fate, using C. elegans to investigate whether there was a genetic program controlling cell death, or apoptosis. In 1986, he identified the first "death genes", ced-3 and ced-4.
H. Robert Horvitz is an American biologist who, with Sydney Brenner and John E. Sulston, won the Nobel Prize for Physiology or Medicine in 2002 for their discoveries about how genes regulate tissue and organ development via a key mechanism called programmed cell death, or apoptosis.
- The Editors of Encyclopaedia Britannica
The Nobel Prize in Physiology or Medicine 2002 was awarded jointly to Sydney Brenner, H. Robert Horvitz and John E. Sulston "for their discoveries concerning genetic regulation of organ development and programmed cell death'"
Oct 8, 2002 · Robert Horvitz (b 1947), Cambridge, MA, USA, has discovered and characterized key genes controlling cell death in C. elegans. He has shown how these genes interact with each other in...
H. Robert Horvitz wants to understand how genes control animal development and behavior. Horvitz and his team use the nematode C. elegans as a model system to identify and analyze evolutionarily conserved molecular and cellular pathways involved in developmental processes such as programmed cell death, developmental timing, cell lineage, and ...
The Nobel Prize in Physiology or Medicine 2002 was awarded jointly to Sydney Brenner, H. Robert Horvitz and John E. Sulston "for their discoveries concerning genetic regulation of organ development and programmed cell death'"
Oct 9, 2024 · He discovered key genes that control cell death (apoptosis) in the microscopic roundworm Caenorhabditis elegans. Nearly identical genes have been identified in other animals, and human counterparts are being pursued as possible therapeutic targets for a variety of human diseases.
